Can Streptococcus mutans cause death?

As part of its ability to adapt to stress, S. mutans activates production of its CSP pheromone. Under high levels of CSP, a small fraction of the population (<10%) undergoes death by suicide (Qi et al., 2005; Perry et al., 2009b). CipB was identified as a major effector in the CSP-induced PCD process.

How is quorum sensing controlled?

Quorum sensing acts by monitoring cell density through chemical signals that allow communication between bacteria in order to regulate the expression of genes involved in virulence, competition, pathogenicity and resistance (Nealson et al., 1970; Hawver et al., 2016; Paul et al., 2018).

When was S mutans discovered?

In 1924, J. Clarke isolated an organism from carious lesions and called it Streptococcus mutans, because he thought the oval shaped cells observed were mutant forms of streptococci (1).

Does everyone have Streptococcus mutans in their mouth?

S. mutans is a bacterium that grows on the teeth of almost everyone’s mouth. W.D.

Does everyone have Streptococcus mutans?

mutans is naturally present in the human oral microbiota, along with at least 25 other species of oral streptococci. The taxonomy of these bacteria remains tentative. Different areas of the oral cavity present different ecological niches, and each species has specific properties for colonizing different oral sites. S.

What is biofilm and quorum sensing?

During the process of biofilm formation microorganisms have the ability to communicate with each other through quorum sensing. Quorum sensing regulates the metabolic activity of planktonic cells, and it can induce microbial biofilm formation and increased virulence.

How do I get rid of S. mutans?

Antibacterial mouthwashes containing chlorhexidine gluconate have been shown to reduce the level of bacteria in the mouth, including lowering the levels of strep mutans, resulting in a lower decay rate.

Who is most likely to get Streptococcus mutans?

The results showed that females (p = 0.006), children with enamel hypoplasia (p = 0.024), children with low birth weights (p = 0.005), those who consume more sweets (p < 0.001), and those with a higher proportion of visible plaque (p = 0.020 and p = 0.041) were more likely to be colonized by S. mutans.