Why do people with anorexia get osteoporosis?

The link between anorexia nervosa and osteoporosis Low estrogen levels contribute to significant losses in bone density. In addition, individuals with anorexia often produce excessive amounts of the adrenal hormone cortisol, which is known to trigger bone loss.

What medications cause osteoporosis?

The medications most commonly associated with osteoporosis include phenytoin, phenobarbital, carbamazepine, and primidone. These antiepileptic drugs (AEDs) are all potent inducers of CYP-450 isoenzymes.

What is the drug of choice for anorexia nervosa?

In addition to SSRI and SNRI drugs, atypical antipsychotics are also used in the treatment of anorexia [5]. Olanzapine, and Quetiapine are one of the most commonly prescribed drugs.

Can osteoporosis caused by anorexia be reversed?

While osteoporosis is a severe problem in patients with eating disorders, bone loss if recognized early can be addressed and potentially reversed. However, once the patient does develop osteoporosis, there is currently no cure for it.

Can malnutrition cause osteoporosis?

Undernutrition, particularly protein undernutrition, contributes to the occurrence of osteoporotic fracture, by lowering bone mass and altering muscle strength. Furthermore, the rate of medical complications after fracture can also be increased by nutritional deficiency.

Do antidepressants cause osteoporosis?

Older men and women who take the most widely used type of antidepressant medication may be at increased risk for bone loss, according to the results of 2 large studies.

Does medication work for anorexia?

Anorexia nervosa. There have been few controlled trials, and most have demonstrated efficacy only for treating comorbid disorders such as depression and obsessive compulsive disorder. There is limited evidence that antidepressants may help maintain weight gain in successfully treated patients.

How long does it take for anorexia to cause osteoporosis?

The loss of bone mineral density (BMD) also takes place rapidly, often occurring within 6 months of disease onset [4] and persisting even after weight recovery [5].